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High tierRandomized Controlled TrialCitation verified

Alirocumab and Cardiovascular Outcomes after Acute Coronary Syndrome

Gregory G Schwartz, P Gabriel Steg, Michael Szarek, Deepak L Bhatt, ODYSSEY OUTCOMES Committees and Investigators - New England Journal of Medicine, 2018

The second large PCSK9-inhibitor outcomes trial. Alirocumab on top of intensive statins reduced recurrent major ischemic events (HR 0.85). Unlike FOURIER, it showed a possible all-cause mortality signal (HR 0.85), though this was not formally significant under the trial's prespecified hierarchical testing - so it is a hypothesis, not a proven mortality benefit.

Key findings

Effect measures

  • Hazard Ratio: 0.85 (primary composite)95% CI 0.78-0.93
  • Hazard Ratio: 0.85 (all-cause mortality; not formally significant)95% CI 0.73-0.98

Why this evidence tier (High)

Risk of bias:
Very large double-blind randomized placebo-controlled trial.
Precision:
Large event count; precise primary estimate. The mortality result is borderline and not formally significant.
Directness:
Hard clinical composite; second independent PCSK9 mechanism.
Consistency:
Event reduction concordant with FOURIER and the statin/ezetimibe evidence.
Funding / COI:
Manufacturer-funded (Sanofi and Regeneron); several authors were employees.

High certainty for event reduction; the all-cause mortality signal is suggestive but not established, so it should not be over-read.

Population:
18,924 patients 1-12 months after an acute coronary syndrome on high-intensity or maximally tolerated statins, randomized to the PCSK9 inhibitor alirocumab vs placebo; median follow-up 2.8 years.
Conflicts of interest:
Industry-funded by Sanofi and Regeneron, which co-develop alirocumab. Several co-authors were employees of the sponsors.
Funding:
Sanofi and Regeneron (co-developers of alirocumab); NCT01663402.

Limitations

  • Mortality signal not formally significant under prespecified testing.
  • Short median follow-up (2.8 years); manufacturer-funded.

How this study is used